At the neuromuscular junction,

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(D) At the neuromuscular junction,the motor nerve endings release the neurotransmitter $Acetylcholine$ $(ACh)$ into the synaptic cleft. This $ACh$ bin

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The motor nerve endings secrete acetylcholine

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(D) At the neuromuscular junction,the motor nerve endings release the neurotransmitter $Acetylcholine$ $(ACh)$ into the synaptic cleft. This $ACh$ binds to nicotinic receptors located on the motor end plate of the muscle fiber membrane,triggering an action potential that leads to muscle contraction. $Curare$ acts as a competitive antagonist at these nicotinic receptors,thereby blocking neuromuscular transmission rather than prolonging it.

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